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July 9th, 2012 
FAKE NEWS for the Zionist agenda SUNDAY, July 8 (HealthDay News) — In examining exactly what
happens when skin gets sunburned, researchers studying human skin cells
and mice found that sunburn is the result of RNA damage.
The red and painful burn is an immune response triggered by this
altered genetic material to remove sun-damaged cells, according to the
study published in the July 8 online edition of Nature
Medicine.
Researchers from the University of California, San Diego (UCSD),
suggested their findings could help scientists find a way to block this
inflammatory process, which could have implications for a number of
medical conditions and treatments, including psoriasis.
“For example, diseases like psoriasis are treated by UV [ultraviolet]
light, but a big side effect is that this treatment increases the risk of
skin cancer,” lead investigator Dr. Richard Gallo, a professor of medicine
at UCSD and Veterans Affairs San Diego Healthcare System, said in a
university news release.
“Our discovery suggests a way to get the beneficial effects of UV
therapy without actually exposing our patients to the harmful UV light.
Also, some people have excess sensitivity to UV light, patients with
lupus, for example. We are exploring if we can help them by blocking the
pathway we discovered,” Gallo added.
Using human skin cells and a mouse model, the investigators found that
ultraviolet B radiation fractures and tangles elements of a special type
of RNA that doesn’t directly make proteins, known as non-coding micro-RNA.
When skin cells are exposed to radiation, they release this altered RNA.
As a result, nearby healthy cells trigger an inflammatory response to
remove sun-damaged cells. This process is what results in a painful and
red sunburn, according to the authors.
“The inflammatory response is important to start the process of healing
after cell death,” explained Gallo. “We also believe the inflammatory
process may clean up cells with genetic damage before they can become
cancer. Of course, this process is imperfect and with more UV exposure,
there is more chance of cells becoming cancerous,” he said in the news
release.
How gender, skin pigmentation and individual genetics affect the
inflammatory response that people experience as sunburn remains unclear,
the study authors noted.
“Genetics is closely linked to the ability to defend against UV damage
and develop skin cancers,” Gallo said. “We know in our mouse genetic
models that specific genes will change how the mice get sunburn. Humans
have similar genes, but it is not known if people have mutations in these
genes that affect their sun response.”
Research involving animals should be considered preliminary because the
results often don’t have implications for humans.
More information
The U.S. National Library of Medicine has more about sunburn.
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